[HN Gopher] The NNT (Number Needed to Treat), explained (2013)
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       The NNT (Number Needed to Treat), explained (2013)
        
       Author : yamrzou
       Score  : 20 points
       Date   : 2022-12-25 11:10 UTC (11 hours ago)
        
 (HTM) web link (www.thennt.com)
 (TXT) w3m dump (www.thennt.com)
        
       | jphoward wrote:
       | NNT is an interesting metric that people were very sanctimonious
       | about when I was at university, but has rightly lost favour.
       | 
       | This is because NNT is actually a terrible metric for any
       | treatment that reduces risk over a long period, including the
       | most important outcomes like mortality.
       | 
       | Imagine a drug that halves your risk of dying of heart disease.
       | Heart disease kills 1 in 4 people in developed countries, and so
       | realistically it would be a no brainer that everyone middle aged
       | or over should take it.
       | 
       | If you did this in a randomised controlled trial, you might
       | recruit 1000 patients, and randomise 500 to this new drug and 500
       | to placebo. If you ran the trial for a year, you might find 1% of
       | the placebo arm died, and only 0.5% in the control arm. This
       | demonstrates a risk reduction of 0.5%, or a NNT of 200.
       | 
       | An NNT, by conventional teaching, is underwhelming.
       | 
       | Is this the right way to think about it? ABSOLUTELY NOT! The high
       | NNT is a consequence of a low event rate, NOT an ineffective
       | therapy. In preventative medicine, where everyone will EVENTUALLY
       | die of something (and 25% the thing you are looking at), it is
       | the RELATIVE risk reduction (50% here, not 0.5%) that matters.
       | 
       | It's different if you have a single one-off therapy, where all
       | the benefit will have been delivered by a certain timepoint, at
       | which you measure the NNT. But for a lot of medicine it is not
       | that simple, and the NNT sucks.
        
         | ggm wrote:
         | But how about if you project it into health economics? If you
         | use it to calculate something like QALY cost?
        
         | nkurz wrote:
         | You've probably thought about this more than I have, but your
         | "take down" of NNT doesn't work for me. Let's use your example.
         | We treat 500 people and observe another 500. 5 of the untreated
         | die, and 2.5 of the treated die (I don't know how either). We
         | observe that the NNT is 200.
         | 
         | And we this is great! If a drug is inexpensive, has minor but
         | not severe side effects, and saves 1/200 people from dying
         | within a year you should absolutely be giving it to everybody.
         | It's a home run. The problem isn't the NNT, it's the
         | expectation of what constitutes a a high or low number.
         | 
         | By contrast, consider another study. We treat 500,000,000
         | people, and observe another 500,000,000. We observe a 50%
         | relative risk reduction among the treated. The drug is
         | inexpensive, and has minor but not severe side effects. Should
         | we create everyone?
         | 
         | I have no idea. It has to depend on how much risk we started
         | with. If the mortality rate is 1% per year among the untreated,
         | then almost definitely yes. But if we observed 2 deaths in the
         | untreated and only 1 from the treated, probably there is a
         | better area to spend our efforts, even if we somehow knew the
         | life we saved just noise.
         | 
         | So yes, the reduction in risk matters, but only in concert with
         | a concept of the baseline risk. By itself, it's not very
         | helpful, but in concert with other data it's great. NNT is a
         | way of expressing this in a single number. There is no apriori
         | reason to believe that 200 is high or low. It may not be
         | perfect, but it's better than just a bare relative risk
         | percentage.
        
         | oldgradstudent wrote:
         | > If you did this in a randomised controlled trial, you might
         | recruit 1000 patients, and randomise 500 to this new drug and
         | 500 to placebo. If you ran the trial for a year, you might find
         | 1% of the placebo arm died, and only 0.5% in the control arm.
         | This demonstrates a risk reduction of 0.5%, or a NNT of 200.
         | 
         | That's because the only thing you've learned during the above
         | trial is that it halves the death rate for one year. You have
         | no idea what happens after that year. It could get better,
         | worse, or the adverse effects would be worse than the cure.
         | 
         | Your assumption that that drug cuts the death rate beyond that
         | single year is an assumption, not a fact.
         | 
         | > An NNT, by conventional teaching, is underwhelming.
         | 
         | As it should be. That's the only thing you can conclude from
         | the trial.
         | 
         | > Is this the right way to think about it? ABSOLUTELY NOT! The
         | high NNT is a consequence of a low event rate, NOT an
         | ineffective therapy.
         | 
         | Yes. The absolute _proven_ benefit is miniscule.
         | 
         | > In preventative medicine, where everyone will EVENTUALLY die
         | of something (and 25% the thing you are looking at), it is the
         | RELATIVE risk reduction (50% here, not 0.5%) that matters.
         | 
         | But you've only had a 1 year trial. Drugs often have long term
         | adverse effects that dwarf the benefit.
         | 
         | As an example, when steroids were first discovered they were
         | viewed as miracle drugs and given in massive doses. It didn't
         | take long to understand the adverse effects were far worse than
         | the symptoms it was treating. Today we are far more careful
         | with steroids.
         | 
         | Similarly with Aspirin, or any number of very useful drugs.
         | 
         | So the question is what gives you the confidence to make any
         | claims beyond what the trial showed?
        
       | yawboakye wrote:
       | bullshit. what about those killed by stopattack? there should at
       | least be 3 distinct effects related to the medication: no effect,
       | positive effect, negative effect. somehow the potential negative
       | effect of the medication is gladly overlooked.
        
         | nickprins wrote:
         | There is a corresponding measure - number needed to harm (NNH).
         | 
         | https://en.m.wikipedia.org/wiki/Number_needed_to_harm
         | 
         | TheNNT also highlights treatments where the evidence for
         | benefit isn't clear (or controversial), e.g. thrombolytics in
         | stroke.
         | 
         | https://www.thennt.com/nnt/thrombolytics-for-stroke/
        
           | yawboakye wrote:
           | it shouldn't be a separate quantity. we should they should be
           | discussed cumulatively at all times.
        
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