[HN Gopher] Newly designed 'smart' insulin could improve type 1 ...
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       Newly designed 'smart' insulin could improve type 1 diabetes
       treatment
        
       Author : lnyan
       Score  : 86 points
       Date   : 2021-08-09 12:09 UTC (1 days ago)
        
 (HTM) web link (www.sciencealert.com)
 (TXT) w3m dump (www.sciencealert.com)
        
       | ruined wrote:
       | is the old insulin free yet
        
         | boublepop wrote:
         | It costs 25$ in wallmart, or if you are uninsured and low
         | income there are patient care programs that will provide it to
         | you for free.
        
           | Trasmatta wrote:
           | Wal-Mart insulin will do in a pinch, but it's pretty garbage
           | compared to the better formulations
        
       | TheBlight wrote:
       | Will they give it away for free in parking lots to anyone who
       | shows up?
        
       | AnthonBerg wrote:
       | This is the paper in question:
       | https://www.pnas.org/content/118/30/e2103518118
       | 
       | Direct PDF link:
       | https://www.pnas.org/content/pnas/118/30/e2103518118.full.pd...
       | 
       | The sciencealert page is a digest of the author's university's
       | digest. The papers themselves are usually much better material.
       | Quite understandable in my experience.
       | 
       | That said, as someone in a relationship with autoimmune diabetes,
       | I hope this work comes to fruition. It's obviously a good idea to
       | try to research and develop this further.
        
       | code_duck wrote:
       | We have been hearing of this in the type 1 diabetes community for
       | some time and it sounds very promising.
       | 
       | As the article explains, the main risk of insulin use is
       | hypoglycemia, which is essentially the result of an insulin
       | overdose. When you dose insulin, you have to carefully match it
       | to the carbohydrates you subsequently consume and the glucose
       | your liver secretes. Too little insulin and you will have high
       | blood glucose, too much and you will suffer low glucose. Highs
       | are uncomfortable and risky long term, but lows are very
       | uncomfortable and immediately dangerous.
       | 
       | For insulin to stop working once your glucose got to 75-80 would
       | be perfect. If the insulin also stayed there and activated as
       | soon as your glucose went up, that might be even better than
       | having a fully functional pancreas.
        
         | cperciva wrote:
         | _For insulin to stop working once your glucose got to 75-80
         | would be perfect._
         | 
         | I'm not so sure about that. Insulin does more than just
         | regulate blood glucose levels; it also affects fat metabolism,
         | potassium levels, and many other things. If the insulin "turned
         | off" completely we could see patients who went into
         | ketoacidosis as a result of fasting and/or exercise.
        
           | code_duck wrote:
           | I wanted to add that one also must know how insulin is dosed
           | to understand this.
           | 
           | Type 1 diabetics typically take two different insulins. One
           | is a 'long acting' that slowly absorbs over 16-24 hours, like
           | what would be called time-release for oral medication. This
           | insulin provides for metabolic function and counteracts the
           | glucose your liver unpredictably and uncontrollably releases
           | at certain times. People with pumps drip regular insulin at a
           | slow rate for the same effect. I assume nothing would change
           | there.
           | 
           | The other type is called 'fast acting' or bolus insulin. This
           | is taken usually in one large dose prior to meals to handle
           | the large rise in blood glucose that follows carbohydrate
           | consumption. The insulin we are discussing here is a
           | replacement for fast acting insulin.
           | 
           | The -ideal- situation for fast acting is for it all to be
           | consumed at the end of digestion. You want fast acting to
           | match 1:1 with the carbohydrates you ingest, every time, with
           | none left to cause hypoglycemia (overdose). other metabolic
           | needs are already handle by the pump drip/long-acting. This
           | innovation would remove the danger of overshooting the
           | insulin dosage and causing hypoglycemia, which is huge.
        
           | code_duck wrote:
           | Yes, your body cannot have zero insulin. You do not need/want
           | more insulin once your glucose is 70-75, though. I'm quite
           | familiar with both hypoglycemia and ketoacidosis, having
           | nearly died of undiagnosed type 1 last Spring.
           | 
           | Ketoacidosis is typically associated with hyperglycemia. It
           | happens when your body has no insulin. There is what's called
           | euglycemic ketoacidosis but it's rare. In typical
           | ketoacidosis, your blood is very high in glucose, but lacking
           | insulin, your cells have no way to obtain it. Since your body
           | cannot get energy out of your blood in the normal fashion it
           | starts to digest ketones, which acidifies the blood and
           | creates many harmful effects.
           | 
           | In the situation we are discussing, when your glucose rises
           | above 75 from food or liver secretion, the novel insulin
           | would start working, preventing ketoacidosis.
        
             | cperciva wrote:
             | _Yes, your body cannot have zero insulin. You do not need
             | /want more insulin once your glucose is 70-75, though. I'm
             | quite familiar with both hypoglycemia and ketoacidosis,
             | having nearly died of undiagnosed type 1 last Spring._
             | 
             | Get another two decades of experience and then we can talk.
             | 
             | It's entirely possible to have blood glucose below 70 mg/dL
             | without having "too much insulin"; healthy adults routinely
             | fall below this level during periods of metabolic
             | starvation. At such times, the body is in a state of
             | ketosis -- this is normal and healthy! -- but as long as
             | there's enough insulin (which there is, in healthy adults)
             | this does not result in acidosis. When A.B. fasted for 382
             | days, his blood glucose levels stabilized around _30_ mg
             | /dL while he remained ambulant.
             | 
             | Ketoacidosis is not so much caused by hyperglycaemia as it
             | is co-occuring as a result of hypoinsulinaemia.
        
               | code_duck wrote:
               | Okay. See you in 20 years then! Just don't raise the bar
               | and tell me it's 40. Currently I have 18 months
               | experience thinking about this.
               | 
               | In the meantime, I am not an expert and am quite willing
               | to be informed. I generally assume that other people in a
               | general discussion audience are not familiar with
               | diabetes treatment or biology at all. If you are, that's
               | great.
               | 
               | I have not heard of healthy states of hypoglycemia
               | before, other than that many people naturally experience
               | it during sleep. I have a friend whose mother suffers
               | from hypoglycemia and she suffers the same ill effects as
               | it how 1 diabetics commonly experience. I am aware that
               | other metabolic causes exist for blood glucose levels to
               | be reduced besides insulin.
               | 
               | Do you mean metabolic ketosis, the type that people try
               | to achieve intentionally through diet? I am aware that
               | states of ketosis exist without acidosis, such as that
               | one. I'm not intimately familiar with the biological
               | details.
               | 
               | Yes, I am aware that ketoacidosis is caused by a complete
               | lack of insulin, and the complete lack of insulin leads
               | to hyperglycemia, not some other causative relationship.
        
               | cperciva wrote:
               | _I have a friend whose mother suffers from hypoglycemia
               | and she suffers the same ill effects as it how 1
               | diabetics commonly experience._
               | 
               | I'm guessing reactive hypoglycaemia? That's caused by the
               | body overproducing insulin in response to meals; it's
               | also believed to be a common prologue to T2D since it
               | "trains" the body to be less sensitive to the
               | (over)produced insulin.
               | 
               | No surprise that hypoglycaemia resulting from an excess
               | of insulin is similar to hypoglycaemia resulting from an
               | excess of insulin!
        
         | AnthonBerg wrote:
         | _For insulin to stop working once your glucose got to 75-80
         | would be perfect. If the insulin also stayed there and
         | activated as soon as your glucose went up, that might be even
         | better than having a fully functional pancreas._
         | 
         | It would be cool!, but I'd go for the pancreas: As far as I
         | know, the pancreas releases insulin in pulses. Each pulse 3-5
         | minutes after the other? The pulses seem to matter. Supposedly
         | the loading-unloading cycle on the insulin receptor seems to
         | help prevent insulin resistance. (It does somehow make
         | intuitive sense to "shake" cells' sensors a bit to help them
         | stay on target.)
         | 
         | Another thing is C-peptide. It's a "byproduct" of the insulin
         | protein folding in the pancreatic cell. I believe C-peptide is
         | now known to be a biologically active control molecule with
         | antiinflammatory properties.
         | 
         | Source: The Wikipedia page on insulin iirc. I an only a layman
         | and may be severely mistaken about all of this.
        
           | code_duck wrote:
           | Of course you need insulin. Just not when your glucose is 75
           | (below 70 is hypoglycemia).
           | 
           | Sure, it's complex.
           | 
           | People with type 1 don't have any insulin production at all,
           | and typically get it in large doses (syringe) that absorb
           | slowly or dripped out from a pump. With no endogenous
           | production, we do not make c-peptide (it's a protein or
           | something cleaved off of pro-insulin in the body's synthesis
           | of insulin. Levels are measured in blood tests to determine
           | your level of insulin production - after honeymoon period,
           | type 1s produce no insulin and therefore no c-peptide).
        
             | AnthonBerg wrote:
             | I was referring specifically to the text as quoted,
             | especially "even better than having a fully functional
             | pancreas" :)
             | 
             | I'm intimately familiar with Type 1 diabetes. I find the
             | nuances of what the pancreas does to be quite fascinating,
             | and the closer we get to understanding what matters and to
             | replicating it, the better.
             | 
             | I believe that one wants the pulsatile secretion as well as
             | C-peptide.
             | 
             | There are some recent results on C-peptide as far as I
             | know. I believe there is still a noteworthy amount of
             | C-peptide after the honeymoon, and I believe it is good to
             | try to prolong the honeymoon as long as possible to retain
             | as much C-peptide functionality as possible.
             | 
             | C-peptide is an active signaling molecule. It makes sense:
             | The body produces a complex and easily identifiable protein
             | chain as a byproduct of the crucial insulin molecule.
             | C-peptide is more stable than insulin itself and remains a
             | bit longer in the body. It stands to reason that the
             | ancient and highly-preserved evolutionary function of
             | insulin metabolism has picked up a good use for C-peptide
             | as a signal carrier on the way.
        
               | code_duck wrote:
               | Ha, yes, it wouldn't -really- be better than your body
               | working as intended. My thoughts were that for regular
               | people, your body has to react to raising glucose by
               | releasing insulin, while with this new insulin, it would
               | already be in your blood.
               | 
               | That pulsing secretion could be achieved with a pump.
               | Maybe they already do it?
               | 
               | I have LADA, which is known for a very long, gradual
               | honeymoon period. I've looked into the studies intended
               | to prolong function but they involve immune suppressants,
               | as far as I can tell, which doesn't seem like a great
               | idea in the Covid age.
        
           | [deleted]
        
       | cletus wrote:
       | Cynical title: Change with questionable benefit extends patent
       | life and thus prices of insulin even further.
       | 
       | To be clear: I don't know a lot about insulin. I'm fortunate
       | enough not to be diabetic. So there may have been useful
       | advancements in insulin. The price of insulin in the US does seem
       | to be awfully high however.
       | 
       | It is a common practice for pharmaceutical companies to alter the
       | active molecule in some way, claim the new version has benefits,
       | reset the patent timeline and stop producing the old version.
       | 
       | EDIT: in case it wasn't clear, if this is actually good for
       | diabetics, that's fantastic. Insulin in the US shouldn't be as
       | expensive as it is. I'm just seen too much profiteering (eg
       | Epipen) by that industry.
        
         | Trasmatta wrote:
         | I am T1 diabetic, and this change, if it works, could
         | absolutely be a game changer.
         | 
         | > So there may have been useful advancements in insulin
         | 
         | There have been massive advancements in insulin over the past
         | couple of decades. Today's long acting and short acting
         | insulins are a massive improvement over the older kinds. The
         | price of insulin in the US is a disaster (when not covered by
         | insurance), but otherwise the quality of insulin treatment is
         | better than it's ever been.
        
           | randcraw wrote:
           | I'm a T2 diabetic, and though I don't (yet) take insulin, I'm
           | pretty sure this could be a very big win for other T2s who
           | do. Dosing insulin is clumsy with an insulin pump, and always
           | hit-or-miss without one (insulin arrives too early or too
           | late or in the wrong amount). The adaptive release of insulin
           | only when glucose levels rise could provide a near-ideal
           | therapy for millions. If it's affordable...
        
         | jcims wrote:
         | I'm not an expert, but have a lot of exposure to this through
         | my kiddo who is a type-1 (insulin deficient) diabetic. This
         | could represent a step function change in her therapy.
         | 
         | Only for folks using the latest and greatest in closed-loop
         | pump therapy is there even an approximation of the pancreas
         | function to elevate insulin levels in response to elevated
         | blood glucose levels, and even then its too slow to be
         | effective. Diabetics that want to closely manage their blood
         | glucose levels have to carefully plan how they eat and how
         | active they are, modulo sickness, periods/etc.
         | 
         | If it was possible to have a latent insulin bolus floating
         | around the bloodstream that could react to gluecose directly,
         | it could provide an important buffer and possibly even a
         | wonderfully passive approach to therapy vs. the ridiculous
         | dance that patients undergo today.
        
         | hooande wrote:
         | This comment is just wrong. This isn't a 'questionable
         | benefit', it's a major change to the fundamental technology.
         | This has nothing to do with price or profits at all. Comparing
         | this theoretical advance to existing insulin is like comparing
         | electric vehicles to gas powered cars. It's a difference in
         | kind, not degree.
         | 
         | To explain, currently your pancreas is measuring your blood
         | sugar and producing more or less insulin to keep it at a safe
         | level (let's call that level "100"). A type 1 diabetic has to
         | do that process manually, measuring their blood sugar with a
         | device and taking enough insulin via needle to keep it at 100.
         | It's possible to overshoot and have blood sugar go far below
         | 100, which can do real damage to cells that need sugar as an
         | energy source. Obviously if you have to measure and adjust
         | several times a day, every day, even a 1% human error rate is
         | going to come up often.
         | 
         | This theoretical insulin would stop working if there wasn't
         | sufficient sugar already in the blood. As another comment said,
         | it would basically stop working if your blood sugar was below
         | 75 or 80. Right now diabetics are at risk from blood sugar
         | being too high OR too low. This would cut out half of that
         | risk, and be life changing for millions of people.
         | 
         | Has nothing to do with price, and as always I must mention that
         | generic insulin is abundant and readily available at low prices
         | to anyone. I don't expect everyone to follow the details of
         | insulin and having diabetes. But it would be nice if medical
         | advancements could be discussed without people conflating the
         | topics with their pet political issues.
        
         | _Microft wrote:
         | I am not a biologist but the proposed behavior of the molecule
         | seems highly beneficial for the patient. It is supposed to only
         | activate in presence of enough glucose instead of always. This
         | is meant to prevent dangerously low blood sugar. They only
         | conducted experiments with fructose so far.
        
         | Someone wrote:
         | If patents were a significant factor in the high prices for
         | insulin in the USA, you would expect prices to be similarly
         | high in all countries that recognize those patents.
         | 
         | They aren't.
         | https://www.rand.org/pubs/research_reports/RRA788-1.html:
         | 
         |  _"Although the ratio of U.S. to other-country prices varied
         | depending on the comparison country and insulin category, U.S.
         | prices were always higher (often five to ten times higher) than
         | those in other countries."_
         | 
         | "Other countries" includes Canada and Germany, so they didn't
         | look at particularly poor countries. The linked PDF says
         | 
         |  _"The average U.S. manufacturer price per standard unit across
         | all insulins was $98.70, compared with $6.94 in Australia,
         | $12.00 in Canada, $7.52 in the United Kingdom, and $8.81 across
         | all non-U.S. OECD countries combined"_
        
           | jellicle wrote:
           | All of those countries set how much they'll pay for insulin.
           | They have price controls. The USA does the opposite; requires
           | governmental medical insurance providers NOT to bargain with
           | pharma companies and instead explicitly requires them to pay
           | whatever price the pharma company sets.
           | 
           | Low prices could come from either a) a robust, competitive
           | market in producing and selling insulin or b) governmental
           | price controls. The USA explicitly chooses not to have
           | either. Other countries typically choose b) backed up by a
           | threat of a) (if you're a pain in the ass over our negotiated
           | price controls, we'll go source a large supply of insulin
           | from somewhere else).
        
             | boublepop wrote:
             | > The USA does the opposite; requires governmental medical
             | insurance providers NOT to bargain with pharma companies
             | and instead explicitly requires them to pay whatever price
             | the pharma company sets.
             | 
             | That's either a gross misunderstanding or deliberate
             | misinformation. There is both price negotiations and
             | significant rebates negotiated by pharmacy benefits
             | managers who will often make 75% of the profit on the sale
             | of the drugs back as rebates. They then distribute that
             | back to insurers and other parties. Sadly those enormous
             | rebates don't seem to reach or benefit the patients.
        
               | jellicle wrote:
               | > > requires governmental medical insurance providers
        
           | RHSeeger wrote:
           | Add to that that the insulins used by a lot of people are
           | over 20 years old and off patent. However, they are biologics
           | and, as such, can't be simply made as a generic. Any
           | "generic" needs to go through testing and approval, which
           | means the generics (like Basaglar is for Lantus) aren't that
           | much cheaper (80% of the cost, I believe).
        
       | ryneandal wrote:
       | I'm a fan of anything to make management of T1/LADA simpler.
       | Didn't realize how much of a chore management is until I was
       | diagnosed with LADA in October of 2019. I feel for all of you
       | that have been dealing with T1 for years/decades. It's
       | exhausting.
        
       | gorb314 wrote:
       | I think this is an interesting research direction.
       | 
       | From my perspective though, being a T1, what I want is an even
       | faster acting insulin than we already have, with a shorter "on"
       | time.
       | 
       | To explain: the shortest acting insulins we have right now take
       | effect about 30 minutes after injection. Then the dose stays
       | active for about 4 hours. So for me to react to a high blood
       | sugar, and until I can be certain that I did not inject too much
       | (nor too little), can take up to 4 hours.
       | 
       | That is almost unbearably long, especially when my blood sugar
       | climbs through the roof, and I am not sure whether it is because
       | my pump line was blocked. Overloading on insulin (called
       | "stacking") to compensate for a crazy high blood sugar can be
       | very dangerous - plus I can't think straight when my sugar is
       | that high. Nothing sucks more than having to sit and wait for a
       | high blood sugar to come down...
       | 
       | Contrast that lagged response time to eating food, especially
       | high GI sugars such as a soft drink, orange juice, or even bread.
       | This can spike my insulin in a matter of minutes.
       | 
       | All this makes matching sugar spikes with insulin response curves
       | very hard; it is a one-side-overdamped-and-other-side-underdamped
       | dynamic system. Any diabetic will tell you how crazy a "roller
       | coaster ride" can be, which is what happens when you
       | overcompensate on either end.
       | 
       | To get back to the topic of the 'smart' insulin: if this research
       | can lead to an insulin that can always be in my bloodstream,
       | ready to act when my blood sugar rises, and stop when my blood
       | sugar falls, and if it can do this quick enough, then that would
       | be a game changer.
        
         | zippergz wrote:
         | Have you tried the inhaled insulin? It takes effect MUCH faster
         | than any injected insulin, does it's job, and is out of your
         | body quickly so also reduces the risk of stacking. In some ways
         | it is not as convenient as a pump, but it works much better in
         | terms of timing.
        
           | zby wrote:
           | How do you take it? I imagine it would be ideal to quickly
           | beat hyperglycemia when you underestimate a meal bolus. But I
           | don't see it replacing a pump (or pens), because it would be
           | difficult to: measure it exactly and keep supplying it
           | continuously.
        
             | danudey wrote:
             | The only inhaled insulin I can find is Afrezza, and WebMD
             | says:
             | 
             | > It's not for diabetes emergencies such as diabetic
             | ketoacidosis (DKA).
             | 
             | So probably not great for this. Seems more like something
             | you take during a meal to keep from going hyperglycemic and
             | not something you take when you're having problems.
             | 
             | https://www.webmd.com/diabetes/inhaled-insulin
        
               | [deleted]
        
             | zippergz wrote:
             | I know people who quite successfully use it in place of a
             | pump, along with a single basal shot from a pen each day.
             | It's just a small inhaler with disposable plastic dose
             | dispensers in various numbers of units. It because it works
             | so quickly it is not a problem to measure accurately. If
             | you use too little, you'll quickly know and can take
             | another. But you are correct that it could not replace
             | basal insulin. I suppose you also could use it as a
             | supplement to a pump and there are probably people who do
             | it, but not in my circle.
        
         | 1123581321 wrote:
         | Fellow T1 here--when you're in that situation you shouldn't sit
         | while your bolus barely keeps up with fast carbs, you should do
         | something physically active to help get your numbers down more
         | quickly.
         | 
         | I share your wish in your last paragraph very much!
        
           | disabled wrote:
           | It's a nice thing to say and all, but not everyone with T1D
           | is abled-bodied. There are other people with T1D who also
           | have to work in an office right after their mealtime so it's
           | also a form of classism. You are also pointing out something
           | that is extremely obvious to people with T1D. You could try
           | to be more thoughtful with your posts and actually post
           | something substantive, useful, and original instead.
           | 
           | Also, your post below about "first world problems" is
           | offensive. First world problems do not exist and it is
           | unbelievably offensive to people suffering in developing
           | countries, like in many places in Africa. Trust me, I know,
           | as I have immediate family who recently lived in Africa on
           | assignment in one of the most corrupt countries in the world
           | for a couple of years. You would not believe how insensitive
           | "first world problems" actually is. I suggest you cut it out
           | of your language, now. Same goes with first world/third
           | world. You need to use "developed countries" and "developing
           | countries".
           | 
           | Please be mindful of your language.
        
           | pixl97 wrote:
           | If only physical activity paid as much as holding a chair
           | down.
        
             | 1123581321 wrote:
             | Yes, it's tough (for the first world.) I've done walking
             | meetings and during COVID some conference calls on the go,
             | but if I ate a bunch of pasta for lunch and didn't take my
             | insulin first, and have typing to do...
        
         | zby wrote:
         | What the article speculates is a way to load up insuline in the
         | blood that would not act if the blood glucose is below let's
         | say 120 - but when it is above - then the insuline opens and
         | acts. This is the fastest possible way of it to act. Even if it
         | degrades and you could not keep much of it constantly in the
         | blood stream - then you could inject it much before the meal
         | and it would just start acting when the blood sugar increases
         | and also you could inject more than enough and not worry about
         | injecting too much of it - because it would never lead to hypo.
         | 
         | This would be the ideal - in a way it would be better than
         | functioning pancreas - because pancreas needs a few minutes
         | before releasing insuline and this would act instantly.
        
           | RHSeeger wrote:
           | It would be nice to have something like this with a several
           | hour window. I currently suffer from the dawn phenomenon, and
           | my blood sugar spikes by 100+ most mornings (starting
           | anywhere between 5am and 7am). It's super frustrating to have
           | to get up every day at 630-7am to check my blood sugar and,
           | if necessary (most days), take some insulin.
        
             | UnpossibleJim wrote:
             | Waking up to the GCM an hour before the alarm every morning
             | is horrible. While maybe not the worst thing about being a
             | T1, I'm still loathe to do it =P
             | 
             | I will say this, though. The CGM (continuous glucose
             | monitor) was one of the best things to help me regulate my
             | blood sugars - even though insurance hates to cover it from
             | time to time (not always - it's weird). As a person who has
             | a tendency to suffer from extreme lows, I'm excited for
             | this new "hinged" insulin, should it make it to market.
        
               | [deleted]
        
         | FigurativeVoid wrote:
         | That's a really great point. I am T1 as well. The improvements
         | I would love is an insulin that is denser and more stable.
         | 
         | If we had insulin that was say.. 1000 units/mL (10x stronger)
         | and didn't degrade as fast, then we could have some really cool
         | and slim devices. You could also have a longer lasting pump
         | site.
        
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