[HN Gopher] Insulin at 100
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       Insulin at 100
        
       Author : crapvoter
       Score  : 62 points
       Date   : 2021-07-19 23:54 UTC (23 hours ago)
        
 (HTM) web link (diatribe.org)
 (TXT) w3m dump (diatribe.org)
        
       | mnw21cam wrote:
       | So, I have a couple of issues with the text.
       | 
       | 1. The article describes the reduction in use of insulin in type
       | 2 diabetes as a bad thing. No. It is a good thing. Type 2
       | diabetes is caused by having too much blood glucose, the body
       | producing more insulin to lower it, fat cells absorbing the
       | glucose, and then this cycle continuing until there is so much
       | energy stored in fat tissue that the amount of insulin required
       | to keep it there is really high, and eventually the body can't
       | produce that much insulin any more. Injecting more insulin
       | stabilises the blood glucose level yes, but it does it by
       | cramming more energy into fat cells that are already full. Diet,
       | exercise, and other glucose-lowering drugs are a much better
       | treatment. Taking insulin just makes you dependent on insulin and
       | potentially even less healthy. Removing the stored energy by some
       | other method (for instance SGLT2-inhibitors make you pee out
       | glucose in your urine[0]) can make you more healthy. If you're up
       | for it and dedicated enough, fasting can often completely reverse
       | type 2 diabetes. The record for the longest fast is 382 days [1]
       | 
       | 2. The article says that "what no one predicted was that GLP-1
       | agonists and SGLT-2 inhibitors have been shown to reduce the risk
       | of both heart and kidney disease". This should be the least
       | surprising thing in the entire world. Most of the management of
       | diabetes these days is to try and get blood glucose levels
       | stable, specifically because of the consequent benefits in
       | reducing the risk of various diseases, including heart and kidney
       | disease. If you have diabetes, your doctor will be trying to
       | reduce these risks, because they are what people with diabetes
       | tend to die from in the long term. That includes giving heart
       | medication. You can live with blood glucose levels out of whack
       | for a long time without noticing anything particularly untoward,
       | but it will be constantly causing damage. So, was anyone
       | surprised that drugs that lower blood glucose can reduce cases of
       | diseases caused by high blood glucose levels? No, they were not -
       | it was obvious.
       | 
       | [0] https://www.fda.gov/drugs/postmarket-drug-safety-
       | information... [1]
       | https://en.wikipedia.org/wiki/Angus_Barbieri%27s_fast
        
         | rdedev wrote:
         | Do we know why the blood glucose gets too high? From what I
         | understand our cells reduce the number of insulin receptors
         | which leads them to absorb less glucose and hence lead to
         | increased blood glucose. But the reason for this insulin
         | resistance is still sort of up for debate. Atleast this is what
         | I understood.
        
           | mnw21cam wrote:
           | There are a few theories out there, and the truth is probably
           | a mixture of them along with some we don't know about.
           | 
           | However, firstly, type 2 diabetes starts out with no
           | significant increase in blood glucose. The reason for this is
           | that the body is very good at self-regulating (homeostasis),
           | so it just produces a bit more insulin to keep glucose levels
           | about right. This happens in the pre-diabetes stage. The
           | insulin tells the fat cells to convert glucose into fat and
           | store it, which causes people to put on weight. Type 2
           | diabetes should be viewed as a disease of too much insulin
           | required, rather than too much glucose present. Yes, there
           | ends up being too much glucose, but the primary problem is
           | that you are giving the body too much energy to store, and it
           | needs to produce too much insulin in order to do that.
           | 
           | This leads into one reason for insulin resistance - the fat
           | cells that are already full need a higher and higher level of
           | insulin to persuade them to keep that energy stored there.
           | Adding more insulin gets them to cram more in, but there is a
           | limit to how much they can.
           | 
           | Another possible reason for insulin resistance is constant
           | high levels of insulin in the blood. Like a drug which you
           | become resistant to if you are constantly taking it, your
           | cells probably get resistant to the insulin. Insulin is
           | secreted in response to high blood glucose and food intake.
           | So there are two things that can cause a constant high level
           | of insulin. The first is if fat cells are full, and they need
           | a high insulin level to stop them dumping the stored energy
           | back into the blood in the form of glucose. The second is if
           | you're constantly snacking. For that reason, it is a real
           | benefit to have a decent long stretch of the day when you
           | don't eat anything. It used to be quite normal to have an
           | evening meal at 6:30pm, and then not eat anything until
           | breakfast the next morning, which gives the body a nice
           | 12-ish-hour session each day of low insulin levels that allow
           | resistance to recover. It's one reason why the 5-2 diet
           | (where you eat normally for five days and then fast or
           | severely diet for two days, every week) is successful.
        
         | bpizzi wrote:
         | I second that. Looks to me that the author is more
         | knowledgeable in T1 than T2, and is almost categorizing the
         | latter as a subspecies of the former, for example:
         | 
         | > Some type 2 patients wrongly associate insulin with personal
         | failure surrounding diet or exercise
         | 
         | Well, I don't want to be that person, but to me the association
         | is tangible.
        
           | mnw21cam wrote:
           | There is a tipping point beyond which type 2 diabetes gets a
           | positive feedback loop, which can make it extremely hard to
           | treat with diet/exercise/drugs. Also, the beta cells in your
           | body that produce insulin can wear out through exhaustion at
           | having to produce so much insulin. Once that has happened,
           | then injecting insulin is necessary.
           | 
           | Some people falsely think this means that your type 2
           | diabetes has progressed to type 1, but that's not true - it
           | is still type 2, just more severe. Type 1 is a completely
           | different disease, but with some similar symptoms.
           | 
           | I quite understand the "personal failure" part of this. You
           | inject insulin if the diet/exercise/drugs weren't good enough
           | to stop the disease progressing.
           | 
           | However it should also be noted that a decent number of
           | people do actually develop type 1 diabetes late in life.
           | Typically they will be misdiagnosed with type 2 diabetes,
           | because doctors tend to assume that diabetes late in life
           | must be type 2. If this happens, a progression on to insulin
           | injections is nigh-inevitable, regardless of how much
           | diet/exercise/drugs they have. There are a few tests that can
           | help distinguish these patients - firstly they tend to be
           | thin, but secondly the auto-antibodies that caused the
           | destruction of the beta cells can often be detected in their
           | blood.
        
             | bpizzi wrote:
             | > but secondly the auto-antibodies that caused the
             | destruction of the beta cells can often be detected in
             | their blood.
             | 
             | That's actually really interesting, I still have got much
             | to learn (and you seems to know that stuff pretty well,
             | thanks for sharing!).
        
       | refurb wrote:
       | Although the article mentions Eli Lilly as the first
       | manufacturers of human insulin made through genetic engineering,
       | that technology actually came from Genentech located in South San
       | Francisco.
       | 
       | Since Genentech was a small biotech company without a lot of
       | money or manufacturing capability they outsourced it to Eli Lilly
       | who was already a leader in the insulin market.
       | 
       | https://www.gene.com/stories/cloning-insulin
        
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       (page generated 2021-07-20 23:03 UTC)