Posts by AlainPetit77@gigaohm.bio
 (DIR) Post #B0ubrAoTIiovWvfVnU by AlainPetit77@gigaohm.bio
       2025-11-23T21:16:24.441814Z
       
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       as well as elsewhere where slaves were kept…
       
 (DIR) Post #B0yjvzMwqx4bkcRREe by AlainPetit77@gigaohm.bio
       2025-12-06T17:23:05.541659Z
       
       1 likes, 0 repeats
       
       Need your help again…Download link for or file?Review of Epidemiology, Pathophysiology, Virology, Clinical Signs, Diagnosis, Prevention, and Treatment of Severe Acute Respiratory Syndrome 2003 (SARS 2003): A Systematic ReviewMarch 2025Coronaviruses 06DOI:10.2174/0126667975349298250307055827Please…
       
 (DIR) Post #B0ylK5kEtrRyQeOPdg by AlainPetit77@gigaohm.bio
       2025-12-06T17:45:00.876761Z
       
       1 likes, 0 repeats
       
       Thanks
       
 (DIR) Post #B0ylnUFmr7Hmq7AKlk by AlainPetit77@gigaohm.bio
       2025-12-06T17:47:33.439249Z
       
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       We present here the clinical features of five infants who died in the neonatal period due to COVID-19, for which the medical records of COVID-positive pregnant women isolated in a third-level neonatal intensive care unit between December 2019 and October 2021 were evaluated retrospectively. Of the five cases, three were preterm, one had a great artery transposition and another underwent surgery for necrotizing enterocolitis. All of the cases experienced a gradual increase in respiratory distress, and none responded to remdesivir, intravenous immunoglobulin or steroid treatments. COVID-19 infection can be fatal in the neonatal period, especially in the presence of an underlying disease or comorbidities such as prematurity and congenital heart disease.https://www.respircase.com/jvi.aspx?un=RCR-58751&volume=
       
 (DIR) Post #B0ypEVpZOqHFPcTtdQ by AlainPetit77@gigaohm.bio
       2025-12-06T18:10:56.014767Z
       
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       Eh Pamela, none of your links offer the entire pdf except this one for 65.00
       
 (DIR) Post #B13mX4YDimpGEeK9wm by AlainPetit77@gigaohm.bio
       2025-12-08T23:57:41.509846Z
       
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       Ian Carroll Tucker Carlson Tim Pool, Yall missed Candace Owens Clinton Foundation DNC cash 1421v2https://stream.housatonic.live/w/ax5pmzSgZJ9gdtofaBxmT3
       
 (DIR) Post #B14wLeMFxXHEVtsd28 by AlainPetit77@gigaohm.bio
       2025-12-09T13:09:48.883510Z
       
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       https://time.com/7338791/childhood-mortality-increasing-gates-foundation/?utm_source=twitter&utm_medium=social&utm_campaign=editorial&utm_content=051225
       
 (DIR) Post #B15T9DY16uoANK3oIK by AlainPetit77@gigaohm.bio
       2025-12-09T23:04:58.577259Z
       
       1 likes, 0 repeats
       
       I have posted this here before…Look at the cover…https://archive.org/details/fruit-from-a-poisonous-tree
       
 (DIR) Post #B1hEOPRhJA5ama7faS by AlainPetit77@gigaohm.bio
       2025-12-28T04:14:49.695818Z
       
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       Just sharing…What they omit is that Adenoviruses were first discovered in 1953, when researchers led by Wallace P. Rowe isolated them from human adenoid tissue at the US National Institutes of Health.They were named adenoviruses because they were initially found in the adenoids, and were soon recognised as causes of respiratory infections, conjunctivitis, and other illnesses.There are more than 80 recognised human types, with roughly 50–60 well-established types, depending on classification updates. They are grouped into seven species: Human adenovirus A–G.They are DNA viruses, so they mutate more slowly than RNA viruses. Adenoviruses change, but they don’t churn. They produce stable types, not fast-moving variants, which is why talking about “adenovirus variants” usually reflects borrowed COVID language rather than virology reality.https://trusttheevidence.substack.com/p/the-christmas-mystery-virus
       
 (DIR) Post #B1mbXN7caQtUuxm5FQ by AlainPetit77@gigaohm.bio
       2025-12-30T18:39:28.660912Z
       
       1 likes, 0 repeats
       
       https://m.youtube.com/watch?v=dbwCLypWRV0
       
 (DIR) Post #B2BHuv3A98phj3xCzI by AlainPetit77@gigaohm.bio
       2026-01-11T16:16:37.010652Z
       
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       California's first significant law regarding mold was the Toxic Mold Protection Act of 2001, which established guidelines for mold presence in residential properties and required landlords to maintain habitable conditions. This law emphasizes the importance of addressing mold issues to protect tenant health and safety.Bacteriophages, or phages, were discovered independently by Frederick W. Twort in 1915 and Félix d’Hérelle in 1917, while the mold that produces penicillin, Penicillium notatum, was discovered by Alexander Fleming in 1928. These discoveries were significant in the development of treatments for bacterial infections and antibiotics.Title: Identification of Allorecognition Loci in Neurospora crassa by Genomics and Evolutionary ApproachesSeries: Molecular Biology and Evolution 2015-may 28 vol. 32 iss. 9Author(s): Zhao, Jiuhai; Gladieux, Pierre; Hutchison, Elizabeth; Bueche, Joanna; Hall, Charles; Perraudeau, Fanny; Glass, N. LouisePublisher:Year: 2015ISBN:Publisher: Caister Academic PressEdited by: Durgadas P. Kasbekar and Kevin McCluskeyCentre for Cellular and Molecular Biology, Hyderabad, India and Fungal Genetics Stock Center, University of Missouri-Kansas City, USA (respectively)Pages: x + 294TimelinePublished: 01 May 2002Neurospora: a model of model microbeshttps://www.nature.com/articles/nrg797
       
 (DIR) Post #B2BI7uIwEIljB6Wfq4 by AlainPetit77@gigaohm.bio
       2026-01-11T15:29:19.891197Z
       
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       The neurospora CYT-18 protein suppresses defects in the phage T4 td intron by stabilizing the catalytically active structure of the intron core
       
 (DIR) Post #B2BIA4pA5YAINqdIfY by AlainPetit77@gigaohm.bio
       2026-01-11T15:08:18.080389Z
       
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       With this inmind, on October 2-4, 1983, we attended a con-ference entitled "Forces Molding the Genome"organized by David Baltimore, the director ofthe newly formed Whitehead Institute at M.I.T.The purpose of the institute is to investigate
       
 (DIR) Post #B2aDeh3oZUFWZmoan2 by AlainPetit77@gigaohm.bio
       2026-01-23T14:07:10.166862Z
       
       1 likes, 0 repeats
       
       https://www.msn.com/en-ca/health/other/suspected-exposure-to-deadly-bleeding-eye-virus-triggers-emergency-at-top-secret-us-lab/ar-AA1UKBGj?ocid=entnewsntp&pc=U531&cvid=697371c5982e400185334ce116c7d8c5&ei=19
       
 (DIR) Post #B348fv1XBiLASbrBqK by AlainPetit77@gigaohm.bio
       2026-02-06T23:39:54.815080Z
       
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       Title: Human DNA polymerase a: predicted functional domains and relationships with viral DNA polymerasesSeries:Author(s): S-e Wang, Teresa (author);Wong, Scott W (author);Korn, David (author)Publisher:Year: Aug 2017 downloadedDownloaded from www.fasebj.org to IP 128.122.230.132. The FASEB Journal Vol.3, No.1ISBN:
       
 (DIR) Post #B37Ri0bTtWREwrGDse by AlainPetit77@gigaohm.bio
       2026-02-08T15:01:54.738971Z
       
       1 likes, 0 repeats
       
       Detecting Pulmonary Oxygen Toxicity Using eNose Technology and Associations between Electronic Nose and Gas Chromatography–Mass Spectrometry DataAbstract: Exposure to oxygen under increased atmospheric pressures can induce pulmonaryoxygen toxicity (POT). Exhaled breath analysis using gas chromatography–mass spectrometry (GC–MS) has revealed that volatile organic compounds (VOCs) are associated with inflammation and lipoperoxidation after hyperbaric–hyperoxic exposure. Electronic nose (eNose) technology would be more suited for the detection of POT, since it is less time and resource consuming. However, it is unknown whether eNose technology can detect POT and whether eNose sensor data can be associated with VOCs of interest. In this randomized cross-over trial, the exhaled breath from divers who had made two dives of 1 h to 192.5 kPa (a depth of 9 m) with either 100% oxygen or compressed air was analyzed, at several time points, using GC–MS and eNose. We used a partial least square discriminant analysis, eNose discriminated oxygen and air dives at 30 min post dive with an area under the receiver operating characteristics curve of 79.9% (95%CI: 61.1–98.6; p = 0.003). A two-way orthogonal partial least square regression (O2PLS) model analysis revealed an R2 of 0.50 between targeted VOCs obtained by GC–MS and eNose sensor data. The contribution of each sensor to the detection of targeted VOCs was also assessed using O2PLS. When all GC–MS fragments were included in the O2PLS model, this resulted in an R2 of 0.08. Thus, eNose could detect POT 30 min post dive, and the correlation between targeted VOCs and eNose data could be assessed using O2PLS.
       
 (DIR) Post #B37Ri67DOxnG2j98XQ by AlainPetit77@gigaohm.bio
       2026-02-08T15:12:02.001718Z
       
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       Mechanistic role of cytochrome P450 (CYP)1B1 in oxygen-mediated toxicity in pulmonary cells: a novel target for prevention of hyperoxic lung injuryABSTRACTSupplemental oxygen, which is routinely administered to preterm infants with pulmonaryinsufficiency, contributes to bronchopulmonary dysplasia (BPD) in these infants. Hyperoxia alsocontributes to the development of acute lung injury (ALI) and acute respiratory distresssyndrome (ARDS) in adults. The mechanisms of oxygen-mediated pulmonary toxicity are notcompletely understood. Recent studies have suggested an important role for cytochrome P450(CYP)1A1/1A2 in the protection against hyperoxic lung injury. The role of CYP1B1 in oxygen-mediated pulmonary toxicity has not been studied. In this investigation, we tested thehypothesis that CYP1B1 plays a mechanistic role in oxygen toxicity in pulmonary cells in vitro.In human bronchial epithelial cell line BEAS-2B, hyperoxic treatment for 1-3 days led todecreased cell viability by about 50-80%. Hyperoxic cytotoxicity was accompanied by anincrease in levels of reactive oxygen species (ROS) by up to 110%, and an increase of TUNEL-positive cells by up to 4.8-fold. Western blot analysis showed hyperoxia to significantly down-regulated CYP1B1 protein level. Also, there was a decrease of CYP1B1 mRNA by up to 38%and Cyp1b1 promoter activity by up to 65%. On the other hand, CYP1B1 siRNA appeared torescue the cell viability under hyperoxia stress, and overexpression of CYP1B1 significantlyattenuated hyperoxic cytotoxicity after 48 h of incubation. In immortalized lung endothelial cellsderived from Cyp1b1-null and wild-type mice, hyperoxia increased caspase 3/7 activities in atime-dependent manner, but endothelial cells lacking the Cyp1b1 gene showed significantlydecreased caspase 3/7 activities after 48 and 72 h of incubation, implying that CYP1B1 mightpromote apoptosis in wild type lung endothelial cells under hyperoxic stress. In conclusion, ourresults support the hypothesis that CYP1B1 plays a mechanistic role in pulmonary oxygentoxicity, and CYP1B1-mediated apoptosis could be one of the mechanisms of oxygen toxicity.Thus, CYP1B1 could be a novel target for preventative and/or therapeutic interventions againstBPD in infants and ALI/ARDS in adults.2
       
 (DIR) Post #B37RiBqm50CvpTV5kG by AlainPetit77@gigaohm.bio
       2026-02-08T15:58:17.710527Z
       
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       A SIMPLE CLINICAL PREDICTIVEINDEX FOR OBJECTIVE ESTIMATES OFMORTALITY IN ACUTE LUNG INJURY(ALI)Jason D. Christie
       
 (DIR) Post #B3C7oGu5B4kFwkTCDo by AlainPetit77@gigaohm.bio
       2026-02-11T00:03:18.282128Z
       
       1 likes, 0 repeats
       
       I have seen my share of potable water pipes…In the great lakes we have a zebra-mussel that is in all water pipes potable or not…Pic below is what potable water pipes look like but remember these zebra-mussels are alive and they actual give you cleaner water…See below pics…
       
 (DIR) Post #B3C7oMFtHQA0XpiBPM by AlainPetit77@gigaohm.bio
       2026-02-11T00:09:28.519542Z
       
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       But…what does your municipality do and use to line their pipes…https://www.tomahawksystem.ca/news-links/25-uncategorized/31-watermain-lining-and-tuberculation-explainedzebra-mussels will live and grow in any type of pipe and it doesn't matter the lining for its natures was of defeating mankind hubris…